HRSB Biennial Reports

Utilization of Breast Cancer Research and Education Funds Reports

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OrganizationInvestigator(s)Link/PubMed ID#Publication/Abstract Title
Icahn School of Medicine at Mount SinaiIrie, Hanna39541980A PROTAC degrader suppresses oncogenic functions of PTK6 inducing apoptosis of breast cancer cells
Icahn School of Medicine at Mount SinaiSilva, Jose34633138miR-424/503 modulates Wnt/β-catenin signaling in the mammary epithelium by targeting LRP6
New York UniversityDavid, GregorybioRxivThe chromatin associated Sin3B is a critical regulator of DNA damage repair through the engagement of the Non-Homologous End Joining pathway
New York UniversityDavid, GregorybioRxivLipocalin 2 upregulation mediates the detrimental effects of therapy-induced senescence in breast cancer cells
New York UniversityDavid, Gregory35500379The origins of cancer cell dormancy
New York UniversityDavid, Gregory37314748Chromatin-associated SIN3B protects cancer cells from genotoxic stress-induced apoptosis and dictates DNA damage repair pathway choice
New York UniversityDavid, Gregory35953598Therapy-induced senescence promotes breast cancer cells plasticity by inducing Lipocalin-2 expression. 
Stony Brook UniversityChung, Jun38201253The Sensitization of Triple-Negative Breast Cancers to Poly ADP Ribose Polymerase Inhibition Independent of BRCA1/2 Mutation Status by Chemically Modified microRNA-489
Stony Brook UniversityChung, Jun37892162Combination Treatment Strategies to Overcome PARP Inhibitor Resistance
Stony Brook UniversityChung, Jun39201393Recent Technologies towards Diagnostic and Therapeutic Applications of Circulating Nucleic Acids in Colorectal Cancers
Albany Medical CollegeBarroso, Margarida39642392Modeling intratumor heterogeneity in breast cancer